APA 6th Edition Galešić, K., Horvatić, I., Batinić, D., Milošević, D., Saraga, M., Glavina Durdov, M. i Galešić Ljubanović, D. (2015). C1Q-NEFROPATIJA: PRIKAZI BOLESNIKA I PREGLED LITERATURE. Liječnički vjesnik, 137 (9-10), 0-0. Preuzeto s https://hrcak.srce.hr/172720
MLA 8th Edition Galešić, Krešimir, et al. "C1Q-NEFROPATIJA: PRIKAZI BOLESNIKA I PREGLED LITERATURE." Liječnički vjesnik, vol. 137, br. 9-10, 2015, str. 0-0. https://hrcak.srce.hr/172720. Citirano 21.10.2019.
Chicago 17th Edition Galešić, Krešimir, Ivica Horvatić, Danica Batinić, Dinko Milošević, Marijan Saraga, Merica Glavina Durdov i Danica Galešić Ljubanović. "C1Q-NEFROPATIJA: PRIKAZI BOLESNIKA I PREGLED LITERATURE." Liječnički vjesnik 137, br. 9-10 (2015): 0-0. https://hrcak.srce.hr/172720
Harvard Galešić, K., et al. (2015). 'C1Q-NEFROPATIJA: PRIKAZI BOLESNIKA I PREGLED LITERATURE', Liječnički vjesnik, 137(9-10), str. 0-0. Preuzeto s: https://hrcak.srce.hr/172720 (Datum pristupa: 21.10.2019.)
Vancouver Galešić K, Horvatić I, Batinić D, Milošević D, Saraga M, Glavina Durdov M i sur. C1Q-NEFROPATIJA: PRIKAZI BOLESNIKA I PREGLED LITERATURE. Liječnički vjesnik [Internet]. 2015 [pristupljeno 21.10.2019.];137(9-10):0-0. Dostupno na: https://hrcak.srce.hr/172720
IEEE K. Galešić, et al., "C1Q-NEFROPATIJA: PRIKAZI BOLESNIKA I PREGLED LITERATURE", Liječnički vjesnik, vol.137, br. 9-10, str. 0-0, 2015. [Online]. Dostupno na: https://hrcak.srce.hr/172720. [Citirano: 21.10.2019.]
Sažetak C1q nephropathy is considered a form of glomerulonephritis, defined by histological findings of dominant C1q immune deposits in renal biopsy. It is a rare disease, most often manifested in children and young adults. The most common clinical manifestation of the disease is nephrotic syndrome, but other renal syndromes could also be found. The cause of the disease is not known, but the immune pathogenesis could be assumed. Often, resistance to glucocorticoid or other immunosuppressive therapy is present, potentially leading to chronic renal insufficiency. We present ten patients with renal biopsy and clinical findings of C1q nephropathy. None of the patients had clinical or serological manifestations of systemic lupus. All patients had normal findings of C3 and C4 components of complement, as well as normal ANF, anti-dsDNA and ANCA antibodies