Case report, case study
https://doi.org/10.21857/yq32oh8gl9
Acute onset of facial nerve palsy associated with Lyme disease
Vita Komen
; Clinic of Neurology, Clinical Hospital Center Rijeka, Rijeka, Croatia
*
Vladimira Vuletic
; Clinic of Neurology, Clinical Hospital Center Rijeka, Rijeka, Croatia
* Corresponding author.
Abstract
Lyme borreliosis (LB) is an anthropozoonosis, caused by different genospecies of the Borrelia burgdorferi sensu lato complex. The infection has been reported in countries throughout the Northern Hemisphere. Spirochetes circulate in small amounts in the blood. They can affect muscles, joints and nervous system. Clinical presentation of Lyme disease can include extreme fatigue, headache, stiff neck, muscle soreness, joint pain, swollen lymph nodes and sore throat. Recognition of an erythema migrans (EM) rash is very important as it is a hallmark symptom of LB. The stages of disease are labeled as early localized, early disseminated, and late disseminated. In areas endemic for Borrelia burgdorferi, Lyme neuroborreliosis (LNB) is estimated to cause 2-25% of peripheral facial palsy cases. Facial palsy in LB can present as part of a triad that includes radicular pain, cranial nerve involvement and lymphocytic pleocytosis in the CSF, or the palsy can be the sole manifestation of the disease. The current guidelines indicate that the diagnosis of LB is based on a two-tier serology at all stages of the infection. The effect of corticosteroid treatment in PFP patients with LB remains uncertain. The course, duration, and success of antibiotic therapy for LB varies according to the disease stage and site of disease manifestations.
Keywords
Lyme disease; facial paralysis; cerebrospinal fluid; serology; corticosteroids
Hrčak ID:
326028
URI
Publication date:
20.12.2024.
Visits: 0 *