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Histological changes in chronic periapical inflammations
; Zavod za dentalnu patologiju Stomatološkog fakulteta, Zagreb
Stanko Stipetić ; Zavod za opću patologiju i patološku anatomiju Stomatološkog fakulteta, Zagreb
Zlatko Leušić ; Centralna zubna poliklinika »M. Milanović«, Zagreb
APA 6th Edition
Najžar-Fleger, D., Stipetić, S. i Leušić, Z. (1972). Histological changes in chronic periapical inflammations. Acta stomatologica Croatica, 7 (1), 0-0. Preuzeto s https://hrcak.srce.hr/108061
MLA 8th Edition
Najžar-Fleger, Dora, et al. "Histological changes in chronic periapical inflammations." Acta stomatologica Croatica, vol. 7, br. 1, 1972, str. 0-0. https://hrcak.srce.hr/108061. Citirano 29.01.2022.
Chicago 17th Edition
Najžar-Fleger, Dora, Stanko Stipetić i Zlatko Leušić. "Histological changes in chronic periapical inflammations." Acta stomatologica Croatica 7, br. 1 (1972): 0-0. https://hrcak.srce.hr/108061
Najžar-Fleger, D., Stipetić, S., i Leušić, Z. (1972). 'Histological changes in chronic periapical inflammations', Acta stomatologica Croatica, 7(1), str. 0-0. Preuzeto s: https://hrcak.srce.hr/108061 (Datum pristupa: 29.01.2022.)
Najžar-Fleger D, Stipetić S, Leušić Z. Histological changes in chronic periapical inflammations. Acta stomatologica Croatica [Internet]. 1972 [pristupljeno 29.01.2022.];7(1). Dostupno na: https://hrcak.srce.hr/108061
D. Najžar-Fleger, S. Stipetić i Z. Leušić, "Histological changes in chronic periapical inflammations", Acta stomatologica Croatica, vol.7, br. 1, str. 0-0, 1972. [Online]. Dostupno na: https://hrcak.srce.hr/108061. [Citirano: 29.01.2022.]
The investigation deals with the histopathological picture of chronic granulation parodontitis in relationship to the organization of the proces in cases of untreated dental pulp. The material submitted to histological investigations was obtained from 213 chronic periapical inflammatory processes after the extraction of teeth whose pulps had not been treated. Teeth were also taken into consideration, the roots of which had been subjected to apicoectomy or periapical curettage immediately following endodontic treatment.In order to follow the course of the organization of the process the 'intensity of the proliferation of the fibrous connective tissue was classified in six groups and the intensity of the inflammatory process in two groups depending on the type of cellular infiltration. In this way 139 samples have been classified in 12 groups (Table 1. and Table 2.) and the course of the fibrous proliferation thus observed. The remaining 74 samples were excluded from this investigation because of proliferation of epithelial cells and the formation of cysts. The results of the investigation presented in Table 2. and graphically depicted on the polygon of relative frequencies (Fig. 1.) show that there exists a tendency towards spontaneous cicatrizationin chronic inflammation of the apical parodontal tissue. Complete cicatrization on the apex of a tooth was found in 19 cases (13.7%) and was most frequently localized in the lateral parts of the upper jaw and in the area of the lower first molar. In this connection the fact must be taken into consideration that a certain percentage of periapical radiolucencies following endodontic treatment will not disappear because of the fact that cicatrization had occurred there even before the endodontic intervention. In cases where cellular infiltration develops in the sense of an exacerbation, proliferation of the connective tissue is scanty or absent altogether and accordingly there is no likelihood of spontaneous healing by the formation of a fibrous scar.
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