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Original scientific paper

https://doi.org/10.26582/k.55.2.7

Metabolic response during high-intensity interval exercise and resting vascular and mitochondrial function in CrossFit participants

Regis C. Pearson orcid id orcid.org/0000-0003-4704-920X ; Integrative Cardiovascular Physiology Lab, Department of Kinesiology, University of Georgia, Athens GA, USA
Alyssa A. Olenick ; Integrative Cardiovascular Physiology Lab, Department of Kinesiology, University of Georgia, Athens GA, USA
Nathan T. Jenkins ; Integrative Cardiovascular Physiology Lab, Department of Kinesiology, University of Georgia, Athens GA, USA


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Abstract

High-intensity functional training (HIFT) can play a major role in preventing cardiometabolic disease. The majority of HIFT interventions incorporate CrossFit (CF) training. We measured aerobic capacity, metabolic response during high-intensity interval exercise (HIIE), resting mitochondrial oxidative capacity, and resting vascular function in adults who participated in CF training (> one year) vs. a sedentary group completing <2 h·wk-1 of structure exercise for > one year (SED). Twenty-one participants were recruited (CF n = 13 vs. SED n = 8). CF participants had a 33.0% greater relative VO2 peak (p<.001) and lower body fat percentage (CF = 18.6 [3.8] vs. SED = 30.3 [8.4]; p<.001). CF participants had higher exercising substrate oxidation when expressed as absolute and body weight relative values (p<.013), but not when expressed relative to lean mass (p>.200). CF participants had greater mitochondrial oxidative capacity (p=.014). There were no differences in large artery function, but CF participants had greater baseline arterial diameter (p=.004) and faster reperfusion following arterial occlusion (p<.05). These data support HIFT programs’ effectiveness in improving fitness, weight status, and metabolic, mitochondrial, and vascular function.

Keywords

high-intensity functional training; CrossFit; metabolism; vascular function

Hrčak ID:

309162

URI

https://hrcak.srce.hr/309162

Publication date:

31.12.2023.

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