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Cerebral glycolysis and glycogenolysis in diazinon treated animals

K. Husain ; Industrial Toxicology Research Centre, Lucknow, India
M.A. Matin ; Industrial Toxicology Research Centre, Lucknow, India

Puni tekst: engleski, pdf (4 MB) str. 29-33 preuzimanja: 120* citiraj
APA 6th Edition
Husain, K. i Matin, M.A. (1986). Cerebral glycolysis and glycogenolysis in diazinon treated animals. Arhiv za higijenu rada i toksikologiju, 37 (1), 29-33. Preuzeto s https://hrcak.srce.hr/153946
MLA 8th Edition
Husain, K. i M.A. Matin. "Cerebral glycolysis and glycogenolysis in diazinon treated animals." Arhiv za higijenu rada i toksikologiju, vol. 37, br. 1, 1986, str. 29-33. https://hrcak.srce.hr/153946. Citirano 27.02.2021.
Chicago 17th Edition
Husain, K. i M.A. Matin. "Cerebral glycolysis and glycogenolysis in diazinon treated animals." Arhiv za higijenu rada i toksikologiju 37, br. 1 (1986): 29-33. https://hrcak.srce.hr/153946
Harvard
Husain, K., i Matin, M.A. (1986). 'Cerebral glycolysis and glycogenolysis in diazinon treated animals', Arhiv za higijenu rada i toksikologiju, 37(1), str. 29-33. Preuzeto s: https://hrcak.srce.hr/153946 (Datum pristupa: 27.02.2021.)
Vancouver
Husain K, Matin MA. Cerebral glycolysis and glycogenolysis in diazinon treated animals. Arh Hig Rada Toksikol. [Internet]. 1986 [pristupljeno 27.02.2021.];37(1):29-33. Dostupno na: https://hrcak.srce.hr/153946
IEEE
K. Husain i M.A. Matin, "Cerebral glycolysis and glycogenolysis in diazinon treated animals", Arhiv za higijenu rada i toksikologiju, vol.37, br. 1, str. 29-33, 1986. [Online]. Dostupno na: https://hrcak.srce.hr/153946. [Citirano: 27.02.2021.]

Sažetak
Treatment with diazinon (40 mg/kg, i. p.) resulted in depletion of glycogen and increased activities of glycogen phosphorylase, phosphoglucomutase, hexokinase and lactate dehydrogenase in the brain of rats. The activity of glucose-o-phosphatase was not significantly changed. The level of lactic acid in the blood was raised, while that of pyruvic was not altered. The induced changes may be due to a compensatory mechanism providing extra energy on account of stimulatory effects in diazinon treated animals.

Hrčak ID: 153946

URI
https://hrcak.srce.hr/153946

[hrvatski]

Posjeta: 298 *