APA 6th Edition Batinac, T., Zamolo, G., Ružić, A. i Peršić, V. (2007). Apoptosis in Skin Cancer Development and Regression. Collegium antropologicum, 31 - Supplement 1 (1), 23-28. Preuzeto s https://hrcak.srce.hr/27416
MLA 8th Edition Batinac, Tanja, et al. "Apoptosis in Skin Cancer Development and Regression." Collegium antropologicum, vol. 31 - Supplement 1, br. 1, 2007, str. 23-28. https://hrcak.srce.hr/27416. Citirano 24.10.2019.
Chicago 17th Edition Batinac, Tanja, Gordana Zamolo, Alen Ružić i Viktor Peršić. "Apoptosis in Skin Cancer Development and Regression." Collegium antropologicum 31 - Supplement 1, br. 1 (2007): 23-28. https://hrcak.srce.hr/27416
Harvard Batinac, T., et al. (2007). 'Apoptosis in Skin Cancer Development and Regression', Collegium antropologicum, 31 - Supplement 1(1), str. 23-28. Preuzeto s: https://hrcak.srce.hr/27416 (Datum pristupa: 24.10.2019.)
Vancouver Batinac T, Zamolo G, Ružić A, Peršić V. Apoptosis in Skin Cancer Development and Regression. Collegium antropologicum [Internet]. 2007 [pristupljeno 24.10.2019.];31 - Supplement 1(1):23-28. Dostupno na: https://hrcak.srce.hr/27416
IEEE T. Batinac, G. Zamolo, A. Ružić i V. Peršić, "Apoptosis in Skin Cancer Development and Regression", Collegium antropologicum, vol.31 - Supplement 1, br. 1, str. 23-28, 2007. [Online]. Dostupno na: https://hrcak.srce.hr/27416. [Citirano: 24.10.2019.]
Sažetak Non-melanoma skin cancers (NMSC) are the most common malignant tumors in white population and their incidence
has been increasing worldwide. Molecular events regulating cell survival, apoptosis, growth arrest as well as cell
differentiation, are important contributors to the overall kinetics of benign and malignant cell growth and play a role in
their development, progression and regression. Failure of these pathways can result in the loss of control over proliferation
and lead to tumor development through the inactivation of tumor suppressor genes or the activation of oncogenes.
Also, immunological mechanisms have been implicated in a phenomenon of tumor progression as well as spontaneous
tumor regression. We have tried to summarize the main events in etiopatogenesis, development, progression and in some
cases skin cancer regression. Further studies are needed to elucidate completely the details of apoptotic control in normal
skin and determine factors resulting in apoptotic disbalance and disease.