Izvorni znanstveni članak

IDIOPATHIC TRIGEMINAL NEURALGIA (ITN): FACTS AND FICTION

Niko Zurak ; School of Medicine, University of Zagreb, Zagreb, Croatia
Darija Mahovic ; School of Medicine, University of Zagreb, Zagreb, Croatia

Sažetak

In this paper the authors present neuroanatomical and neurophysiological arguments against the microvascular compression in
the root entry zone of trigeminal nerve nerve as an ethiopathogenetic factor of ITN. Clinical experience has proven that compre ssion
of mixed sensorymotor nerve (peripheral or central one), cannot provoke paroxysmal neuralgic pain.
The authors conclude that the well known fact that dental pulp has only pain sensory modality brings up the question what might
be consequence of tooth extraction where neural fibers are broken in th e innervation areas of maxillar and mandibular nerve. Th e
answer could be only one. If exclusive algophoric deafferentation hypersensitivity after tooth extraction exceeds a certain thr eshold,
patients will experience paroxysmal neuralgic pain. Broken neural fibers develop pathological ephaptic communication with other
trigeminal sensory modalities through supraspinal central structures responsible for the transmision of dental pulp pain. This can
explain trigger phenomena and latency between the touching of circumoral areas and onset of neuralgic paroxysm, which is a
central epileptic phenomenon. In conclusion, the so-called idiopathic trigeminal neuralgia (ITN) is the expression of algophori c
deafferentation hypersensitivity after tooth extraction

Ključne riječi

idiopathic trigeminal neuralgia; tooth extraction; algophoric deafferentation hypersensitivity; epileptic neuralgic phenomenon; ephaptic neural communication

Hrčak ID:

269446

URI

https://hrcak.srce.hr/269446

Datum izdavanja:

21.12.2019.

Posjeta: 372 *