Review article
A hypothesis of the possible immunological mechanisms behind the chronic gastritis and peptic ulcerations associated with the campylobacter pylori infection
Sven Kurbel
Beatrica Kurbel
Full text: croatian pdf 1.361 Kb
page 21-23
downloads: 162
cite
APA 6th Edition
Kurbel, S. & Kurbel, B. (1989). A hypothesis of the possible immunological mechanisms behind the chronic gastritis and peptic ulcerations associated with the campylobacter pylori infection. Medicinski vjesnik, 21 ((1-2)), 21-23. Retrieved from https://hrcak.srce.hr/196766
MLA 8th Edition
Kurbel, Sven and Beatrica Kurbel. "A hypothesis of the possible immunological mechanisms behind the chronic gastritis and peptic ulcerations associated with the campylobacter pylori infection." Medicinski vjesnik, vol. 21, no. (1-2), 1989, pp. 21-23. https://hrcak.srce.hr/196766. Accessed 23 May 2024.
Chicago 17th Edition
Kurbel, Sven and Beatrica Kurbel. "A hypothesis of the possible immunological mechanisms behind the chronic gastritis and peptic ulcerations associated with the campylobacter pylori infection." Medicinski vjesnik 21, no. (1-2) (1989): 21-23. https://hrcak.srce.hr/196766
Harvard
Kurbel, S., and Kurbel, B. (1989). 'A hypothesis of the possible immunological mechanisms behind the chronic gastritis and peptic ulcerations associated with the campylobacter pylori infection', Medicinski vjesnik, 21((1-2)), pp. 21-23. Available at: https://hrcak.srce.hr/196766 (Accessed 23 May 2024)
Vancouver
Kurbel S, Kurbel B. A hypothesis of the possible immunological mechanisms behind the chronic gastritis and peptic ulcerations associated with the campylobacter pylori infection. Medicinski vjesnik [Internet]. 1989 [cited 2024 May 23];21((1-2)):21-23. Available from: https://hrcak.srce.hr/196766
IEEE
S. Kurbel and B. Kurbel, "A hypothesis of the possible immunological mechanisms behind the chronic gastritis and peptic ulcerations associated with the campylobacter pylori infection", Medicinski vjesnik, vol.21, no. (1-2), pp. 21-23, 1989. [Online]. Available: https://hrcak.srce.hr/196766. [Accessed: 23 May 2024]
Full text: english pdf 1.361 Kb
page 21-23
downloads: 84
cite
APA 6th Edition
Kurbel, S. & Kurbel, B. (1989). A hypothesis of the possible immunological mechanisms behind the chronic gastritis and peptic ulcerations associated with the campylobacter pylori infection. Medicinski vjesnik, 21 ((1-2)), 21-23. Retrieved from https://hrcak.srce.hr/196766
MLA 8th Edition
Kurbel, Sven and Beatrica Kurbel. "A hypothesis of the possible immunological mechanisms behind the chronic gastritis and peptic ulcerations associated with the campylobacter pylori infection." Medicinski vjesnik, vol. 21, no. (1-2), 1989, pp. 21-23. https://hrcak.srce.hr/196766. Accessed 23 May 2024.
Chicago 17th Edition
Kurbel, Sven and Beatrica Kurbel. "A hypothesis of the possible immunological mechanisms behind the chronic gastritis and peptic ulcerations associated with the campylobacter pylori infection." Medicinski vjesnik 21, no. (1-2) (1989): 21-23. https://hrcak.srce.hr/196766
Harvard
Kurbel, S., and Kurbel, B. (1989). 'A hypothesis of the possible immunological mechanisms behind the chronic gastritis and peptic ulcerations associated with the campylobacter pylori infection', Medicinski vjesnik, 21((1-2)), pp. 21-23. Available at: https://hrcak.srce.hr/196766 (Accessed 23 May 2024)
Vancouver
Kurbel S, Kurbel B. A hypothesis of the possible immunological mechanisms behind the chronic gastritis and peptic ulcerations associated with the campylobacter pylori infection. Medicinski vjesnik [Internet]. 1989 [cited 2024 May 23];21((1-2)):21-23. Available from: https://hrcak.srce.hr/196766
IEEE
S. Kurbel and B. Kurbel, "A hypothesis of the possible immunological mechanisms behind the chronic gastritis and peptic ulcerations associated with the campylobacter pylori infection", Medicinski vjesnik, vol.21, no. (1-2), pp. 21-23, 1989. [Online]. Available: https://hrcak.srce.hr/196766. [Accessed: 23 May 2024]
Abstract
A short review of the data regarding the presence of Campylobacter pylori in the peptic ulcer patients and those with chronic gastritis has been presented together with the data regarding the presence and functions of the gastric mucosal mast cells and IgE molecules at their surface. A speculation is made that the physiological role of the IgE/mast cell system in the gastric mucosa, beside bringing the immunocompetent cells to the place of infection, consists also in producing a local tide in the gastric acid secretion that would facilitate pathogen elimination. Chronic infection with C. pylori and other pathogens could lead to the permanent activation of this system resulting in the chronic gastritis or spots of hyperacidity that might turn into ulcers. Findings of the humoral response to C. pylori in ulcer patients support the presented hypothesis. Further studies of the total and specific IgE content in the gastric mucosa of ulcer patients and experimental animals are required to test its validity.
Keywords
Campylobacter pylori; gastritis; IgE; infection; peptic ulcer
Hrčak ID:
196766
URI
https://hrcak.srce.hr/196766
Publication date:
1.12.1989.
Article data in other languages:
croatian
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